Food allergy may be at least partly genetically determined. The risk of Peanut.Allergy, for case in point, with reference to 10 times higher in a child by means of a sibling as well is allergic to peanuts than the wide-ranging population, but to date no precise genes have been identified. So even for non.IgE mediated food allergy, in which there was a large ethnic difference in the incidence and family, with the predominance of male Caucasians.
In families where more than one parent has allergies, the risk to children have food allergies is very high, even if you can not predict food allergies than other types of allergy.
∙ 80% chance of allergy if both parents are atopic manifestations associated with several (eg, asthma and food allergy combined)
∙ 40 -60% chance of allergy if both parents are atopic
∙ 25 to 40% chance of allergy when only one parent is affected by allergy
∙ 20 to 25% chance of allergy when a brother is allergic
Location of events
The T cells reside permanently in the target organs may explain why some foods are localized allergic diseases, and have no systemic features as in the case of atopic dermatitis and eosinophilic esophagitis. The events and the most prevalent of food allergies are the result of a complex, not entirely known, mechanism of relationships between host, environment, food and genetic causes, so what, this, among other things that makes difficult the development of causal therapies.